{"id":12782,"date":"2025-07-25T10:00:22","date_gmt":"2025-07-25T14:00:22","guid":{"rendered":"https:\/\/www.rigicon.com\/glossary-term\/hyperoxaluria\/"},"modified":"2025-07-25T10:00:22","modified_gmt":"2025-07-25T14:00:22","slug":"hiperoxaluria-pt-br","status":"publish","type":"glossary-term","link":"https:\/\/www.rigicon.com\/pt-br\/glossary-term\/hiperoxaluria-pt-br\/","title":{"rendered":"Hiperoxal\u00faria"},"content":{"rendered":"","protected":false},"excerpt":{"rendered":"","protected":false},"featured_media":0,"template":"","glossary-category":[126],"glossary-product":[],"class_list":["post-12782","glossary-term","type-glossary-term","status-publish","hentry","glossary-category-urological-conditions"],"acf":{"gt-definition":"<p>A hiperoxal\u00faria \u00e9 um dist\u00farbio metab\u00f3lico caracterizado pela excre\u00e7\u00e3o excessiva de oxalato na urina, normalmente definida como uma excre\u00e7\u00e3o urin\u00e1ria de oxalato superior a 40 mg (440 \u03bcmol) por 24 horas.<sup class=\"citation\">1<\/sup> Oxalato \u00e9 uma subst\u00e2ncia qu\u00edmica natural produzida pelo corpo e encontrada em certos alimentos que, quando presente em altas concentra\u00e7\u00f5es, forma cristais com c\u00e1lcio nos rins.<sup class=\"citation\">2<\/sup> Esses c\u00e1lcio cristais de oxalato podem levar \u00e0 forma\u00e7\u00e3o de c\u00e1lculos renais, nefrocalcinose (dep\u00f3sitos de c\u00e1lcio no tecido renal) e, em casos graves, danos e insufici\u00eancia renal.<sup class=\"citation\">3<\/sup> A hiperoxal\u00faria pode ser classificada em formas prim\u00e1rias (dist\u00farbios gen\u00e9ticos herdados do metabolismo do glioxilato) e formas secund\u00e1rias (resultantes do aumento da ingest\u00e3o alimentar ou da absor\u00e7\u00e3o intestinal de oxalato).<sup class=\"citation\">4<\/sup><\/p>\n<p>A hiperoxal\u00faria \u00e9 a excre\u00e7\u00e3o urin\u00e1ria de oxalato superior a 40-45 mg\/dia, sendo um fator de risco importante para a forma\u00e7\u00e3o de c\u00e1lculos renais de oxalato de c\u00e1lcio. Pode ser classificada como prim\u00e1ria (gen\u00e9tica, rara, por defici\u00eancia enzim\u00e1tica hep\u00e1tica), ent\u00e9rica (secund\u00e1ria a m\u00e1 absor\u00e7\u00e3o intestinal, como ap\u00f3s bypass g\u00e1strico) ou diet\u00e9tica (ingesta elevada de alimentos ricos em oxalato).<\/p>","gt-synonyms":"Oxal\u00faria, Hiperoxalose (quando se refere \u00e0 deposi\u00e7\u00e3o sist\u00eamica de oxalato), Hiperoxal\u00faria prim\u00e1ria (PH1, PH2, PH3 para formas gen\u00e9ticas), Hiperoxal\u00faria ent\u00e9rica, Hiperoxal\u00faria diet\u00e9tica, Hiperoxal\u00faria idiop\u00e1tica","gt-citation":"[1] Shah A, Leslie SW, Ramakrishnan S. Hyperoxaluria. StatPearls. 2024 Mar 4. PMID: 35015037. https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK558987\/\n\n[2] Groothoff JW, Metry E, Deesker L, et al. Clinical practice recommendations for primary hyperoxaluria: an expert consensus statement from ERKNet and OxalEurope. Nat Rev Nephrol. 2023;19:194-211. DOI: 10.1038\/s41581-022-00661-1\n\n[3] Bhasin B, \u00dcrekli HM, Atta MG. Primary and secondary hyperoxaluria: Understanding the enigma. World J Nephrol. 2015;4(2):235-244. DOI: 10.5527\/wjn.v4.i2.235\n\n[4] Demoulin N, Aydin S, Gillion V, et al. Pathophysiology and Management of Hyperoxaluria and Oxalate Nephropathy: A Review. Am J Kidney Dis. 2022;79(6):885-897. DOI: 10.1053\/j.ajkd.2021.09.014","gt-clinical-context":"<p>A hiperoxal\u00faria contribui significativamente para a nefrolit\u00edase (c\u00e1lculos renais), sendo o oxalato urin\u00e1rio o promotor qu\u00edmico mais forte da forma\u00e7\u00e3o de c\u00e1lculos renais.<sup class=\"citation\">1<\/sup> A apresenta\u00e7\u00e3o cl\u00ednica varia de acordo com o tipo e a gravidade da hiperoxal\u00faria, mas as manifesta\u00e7\u00f5es comuns incluem c\u00e1lculos renais recorrentes, hemat\u00faria (sangue na urina), infec\u00e7\u00f5es do trato urin\u00e1rio e dor no flanco.<sup class=\"citation\">2<\/sup><\/p>\n \n <p>A hiperoxal\u00faria prim\u00e1ria (HP) \u00e9 uma doen\u00e7a heredit\u00e1ria rara com tr\u00eas tipos principais (PH1, PH2, PH3), cada um causado por diferentes muta\u00e7\u00f5es gen\u00e9ticas que afetam as enzimas hep\u00e1ticas envolvidas no metabolismo do glioxilato.<sup class=\"citation\">3<\/sup> PH1, a forma mais grave, \u00e9 causada por muta\u00e7\u00f5es no gene AGXT, resultando em atividade deficiente da alanina-glioxilato aminotransferase.<sup class=\"citation\">4<\/sup> Sem tratamento, a PH1 geralmente progride para doen\u00e7a renal em est\u00e1gio terminal (DRT) na inf\u00e2ncia ou no in\u00edcio da idade adulta.<sup class=\"citation\">5<\/sup><\/p>\n \n <p>A hiperoxal\u00faria secund\u00e1ria inclui hiperoxal\u00faria ent\u00e9rica (resultante de doen\u00e7as intestinais como a doen\u00e7a de Crohn ou ap\u00f3s cirurgia bari\u00e1trica), hiperoxal\u00faria diet\u00e9tica (devido ao consumo excessivo de alimentos ricos em oxalato) e hiperoxal\u00faria idiop\u00e1tica.<sup class=\"citation\">6<\/sup><\/p>\n \n <p>As estrat\u00e9gias de manejo dependem da causa subjacente e da gravidade, mas geralmente incluem aumento da ingest\u00e3o de l\u00edquidos (visando> 3L de produ\u00e7\u00e3o de urina diariamente), restri\u00e7\u00e3o diet\u00e9tica de oxalato, suplementa\u00e7\u00e3o de c\u00e1lcio com refei\u00e7\u00f5es, alcaliniza\u00e7\u00e3o urin\u00e1ria com citrato de pot\u00e1ssio e, em casos graves, tratamentos especializados como piridoxina (vitamina B6) para pacientes com PH1 responsivos. produ\u00e7\u00e3o.<sup class=\"citation\">8<\/sup> Em casos avan\u00e7ados com insufici\u00eancia renal, di\u00e1lise intensiva e transplante combinado de f\u00edgado e rim podem ser necess\u00e1rios, especialmente para HP1.<sup class=\"citation\">9<\/sup><\/p>\n<p>O diagn\u00f3stico requer coleta de urina de 24 horas. A hiperoxal\u00faria prim\u00e1ria \u00e9 a forma mais grave, podendo levar \u00e0 oxalose sist\u00eamica e insufici\u00eancia renal. A EAU recomenda: redu\u00e7\u00e3o de alimentos ricos em oxalato (espinafre, ruibarbo, chocolate, nozes), ingesta adequada de c\u00e1lcio com as refei\u00e7\u00f5es (que se liga ao oxalato no intestino), hidrata\u00e7\u00e3o > 2,5 L\/dia e suplementa\u00e7\u00e3o de citrato de pot\u00e1ssio. A hiperoxal\u00faria ent\u00e9rica responde ao tratamento da causa intestinal e citrato.<\/p>","gt-related-evidence":null,"gt-featured":true,"gt-ai-referenced":false,"gt-view-count":82},"yoast_head":"<!-- This site is optimized with the Yoast SEO Premium plugin v27.9 (Yoast SEO v27.9) - https:\/\/yoast.com\/product\/yoast-seo-premium-wordpress\/ -->\n<title>Hiperoxal\u00faria: Excesso de Oxalato e C\u00e1lculos Renais | Rigicon<\/title>\n<meta name=\"description\" content=\"Conhe\u00e7a a hiperoxal\u00faria, excre\u00e7\u00e3o excessiva de oxalato urin\u00e1rio associada a c\u00e1lculos renais de oxalato de c\u00e1lcio. Tipos, diagn\u00f3stico e manejo. 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