{"id":11889,"date":"2025-07-25T09:59:57","date_gmt":"2025-07-25T13:59:57","guid":{"rendered":"https:\/\/www.rigicon.com\/glossary-term\/hypercalciuria\/"},"modified":"2025-07-25T09:59:57","modified_gmt":"2025-07-25T13:59:57","slug":"hyperkalziurie","status":"publish","type":"glossary-term","link":"https:\/\/www.rigicon.com\/de\/glossary-term\/hyperkalziurie\/","title":{"rendered":"Hyperkalziurie"},"content":{"rendered":"","protected":false},"excerpt":{"rendered":"","protected":false},"featured_media":0,"template":"","glossary-category":[126],"glossary-product":[],"class_list":["post-11889","glossary-term","type-glossary-term","status-publish","hentry","glossary-category-urological-conditions"],"acf":{"gt-definition":"Hyperkalziurie ist eine Stoffwechselerkrankung, die durch eine \u00fcberm\u00e4\u00dfige Ausscheidung von Kalzium im Urin gekennzeichnet ist und das Risiko der Bildung von kalziumbasierten Nierensteinen erheblich erh\u00f6ht.<span class=\"citation\">1<\/span> Sie gilt als der h\u00e4ufigste erkennbare metabolische Risikofaktor f\u00fcr Kalziumnephrolithiasis und macht etwa 50 % der F\u00e4lle aus.<span class=\"citation\">1<\/span> Die Erkrankung kann auf verschiedene Arten definiert werden: traditionell als Kalziumausscheidung im Urin von mehr als 275 mg\/Tag bei M\u00e4nnern und 250 mg\/Tag mg\/Tag bei Frauen; als Ausscheidung von mehr als 4 mg\/kg K\u00f6rpergewicht pro Tag; oder als Kalziumkonzentration im Urin \u00fcber 200 mg\/L.<span class=\"citation\">2<\/span> Hyperkalziurie tr\u00e4gt auch zur Demineralisierung der Knochen bei, was zu einer verringerten Knochenmineraldichte, Osteopenie und m\u00f6glicherweise Osteoporose f\u00fchrt.<span class=\"citation\">3<\/span> Die Pathophysiologie variiert je nach Typ, wobei absorbierende Hyperkalziurie (erh\u00f6hte intestinale Kalziumabsorption) am h\u00e4ufigsten vorkommt, gefolgt von Nierenleck-Hyperkalziurie (defektes renales tubul\u00e4res Kalzium). Reabsorption), renale Phosphatleck-Hyperkalzurie und resorptive Hyperkalzurie (typischerweise aufgrund von Hyperparathyreoidismus).<span class=\"citation\">1<\/span>","gt-synonyms":"Idiopathische Hyperkalziurie, Kalzium\u00fcberausscheidung, \u00fcberm\u00e4\u00dfiger Kalziumaustritt im Urin, renales Kalziumleck, absorbierende Hyperkalziurie, resorptive Hyperkalziurie, renale Phosphatleck-Hyperkalziurie","gt-citation":"[1] Leslie SW, Sajjad H. Hypercalciuria. [Aktualisiert 2024 Feb 12]. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2024 Jan-. Verf\u00fcgbar unter: https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK448183\/\n\n[2] Coe FL, Worcester EM, Evan AP. Idiopathic hypercalciuria and formation of calcium renal stones. Nat Rev Nephrol. 2016 Sep;12(9):519-33. doi: 10.1038\/nrneph.2016.101. Epub 2016 Jul 25. PMID: 27452364; PMCID: PMC5837277.\n\n[3] Hypercalciuria: Practice Essentials, Background, Pathophysiology. Medscape. Updated: May 19, 2023. Verf\u00fcgbar unter: https:\/\/emedicine.medscape.com\/article\/2182757-overview","gt-clinical-context":"Hyperkalziurie ist in zwei Hauptkontexten klinisch bedeutsam: als Hauptrisikofaktor f\u00fcr die Bildung von Nierensteinen und als Mitwirkender bei der Knochendemineralisierung.<span class=\"citation\">1<\/span> Nierensteine auf Kalziumbasis (Kalziumoxalat und Kalziumphosphat) machen etwa 85 % aller Nierensteine aus, wobei Hyperkalziurie die h\u00e4ufigste Ursache f\u00fcr idiopathische Nierensteine auf Kalziumbasis ist.<span class=\"citation\">1<\/span> Die Patientenauswahl f\u00fcr die Beurteilung umfasst typischerweise Patienten mit wiederkehrenden Nierensteinen, unerkl\u00e4rliche H\u00e4maturie oder Probleme mit der Knochendichte.<span class=\"citation\">3<\/span> Zu den diagnostischen Verfahren geh\u00f6rt die 24-Stunden-Urinsammlung zur Messung der Kalziumausscheidung, des Serumkalziumspiegels und des Parathormonspiegels sowie manchmal Kalziumbelastungstests, um die spezifische Art der Hyperkalziurie zu bestimmen.<span class=\"citation\">2<\/span> Die Behandlungsans\u00e4tze variieren je nach Typ, umfassen jedoch im Allgemeinen Ern\u00e4hrungsumstellungen (moderate Kalziumaufnahme von 600\u2013800 mg\/Tag, reduzierte Natrium- und tierische Proteine, erh\u00f6hte Fl\u00fcssigkeitsaufnahme) und Pharmakologische Interventionen wie Thiaziddiuretika, die die renale Kalziumreabsorption verbessern.<span class=\"citation\">3<\/span> Zu den erwarteten Ergebnissen geh\u00f6ren eine verringerte Steinbildung und eine Stabilisierung der Knochenmineraldichte, obwohl typischerweise eine langfristige Behandlung erforderlich ist.<span class=\"citation\">1<\/span> Bei p\u00e4diatrischen Patienten kann sich Hyperkalziurie auch ohne Steinbildung mit H\u00e4maturie, Bauchschmerzen und Harnbeschwerden manifestieren, was altersspezifische diagnostische Kriterien erfordert.<span 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